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Analysis of secondary variables confirmed that participants on semaglutide experienced greater enhancements in fasting and postprandial glucose, insulin secretion and BMI than those on dapagliflozin (Table two). In distinction, dapagliflozin treatment method enhanced the homoeostasis design evaluation two estimate of insulin resistance (HOMA2-IR) as opposed with semaglutide procedure. When the results disaggregated by clusters were being analysed, semaglutide was identified to cause greater reductions in fasting glucose, glucose at 2 h on the oral glucose tolerance test (OGTT) and common glucose focus for the duration of continuous monitoring, as well as a better time in range in participants with SIDD than in These with SIRD (Table two, Fig. 2 and Supplementary Table four). A tendency for enhanced nonalcoholic fatty liver sickness (NAFLD) liver Unwanted fat rating during the SIRD team as opposed Along with the SIDD group in response to semaglutide was also noticed (Prolonged Facts Fig.
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